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AB5790

Sigma-Aldrich

Anti-RBP-Jk Antibody

Chemicon®, from rabbit

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About This Item

UNSPSC Code:
12352203
eCl@ss:
32160702
NACRES:
NA.41

biological source

rabbit

Quality Level

100

antibody form

affinity purified immunoglobulin

antibody product type

primary antibodies

clone

polyclonal

purified by

affinity chromatography

species reactivity

mouse, human

manufacturer/tradename

Chemicon®

technique(s)

ChIP: suitable
immunohistochemistry: suitable
western blot: suitable

NCBI accession no.

NM_005349.2
NM_015874.3
NM_203283.1
NM_203284.1

UniProt accession no.

Q06330

shipped in

dry ice

target post-translational modification

unmodified

Gene Information

human ... RBPJ(3516)

Specificity

Recognizes RBP-Jk / RBP-L transcription factor.

Immunogen

Synthetic peptide, amino acids 194-206 from human RBP-Jk.

Application

Anti-RBP-Jk Antibody is an antibody against RBP-Jk for use in WB, IH.

Legal Information

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

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Storage Class Code

12 - Non Combustible Liquids

WGK

WGK 2

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Eric W Brunskill et al.
Journal of the American Society of Nephrology : JASN, 22(12), 2213-2225 (2011-10-29)
Renin-expressing cells modulate BP, fluid-electrolyte homeostasis, and kidney development, but remarkably little is known regarding the genetic regulatory network that governs the identity of these cells. Here we compared the gene expression profiles of renin cells with most cells in
Histone demethylase KDM5A is an integral part of the core Notch-RBP-J repressor complex.
Liefke, R; Oswald, F; Alvarado, C; Ferres-Marco, D; Mittler, G; Rodriguez, P; Dominguez et al.
Genes & Development null
Marie Cibois et al.
RNA (New York, N.Y.), 16(1), 10-15 (2009-11-26)
Targeted inactivations of RNA-binding proteins (RNA-BPs) can lead to huge phenotypical defects. These defects are due to the deregulation of certain mRNAs. However, we generally do not know, among the hundreds of mRNAs that are normally controlled by one RNA-BP
Olga Gonzalez-Lopez et al.
The Journal of biological chemistry, 294(35), 13073-13092 (2019-07-17)
Reactivation of Kaposi's sarcoma-associated herpesvirus (KSHV) from latency requires the viral transactivator Rta to contact the host protein Jκ recombination signal-binding protein (RBP-Jκ or CSL). RBP-Jκ normally binds DNA sequence-specifically to determine the transcriptional targets of the Notch-signaling pathway, yet
Shinya Ohashi et al.
Gastroenterology, 139(6), 2113-2123 (2010-08-31)
The Notch receptor family regulates cell fate through cell-cell communication. CSL (CBF-1/RBP-jκ, Su(H), Lag-1) drives canonical Notch-mediated gene transcription during cell lineage specification, differentiation, and proliferation in the hematopoietic system, the intestine, the pancreas, and the skin. However, the functional
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