735949
Gadolinium(III) chloride
anhydrous, beads, −10 mesh, 99.99% trace metals basis
Synonym(s):
Gadolinium trichloride
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About This Item
Recommended Products
grade
anhydrous
Assay
99.99% trace metals basis
form
beads
particle size
−10 mesh
SMILES string
Cl[Gd](Cl)Cl
InChI
1S/3ClH.Gd/h3*1H;/q;;;+3/p-3
InChI key
MEANOSLIBWSCIT-UHFFFAOYSA-K
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Application
Gadolinium Chloride is a commonly used precursor for the formation of MRI contrast agent and can be used as a Diels-Alder catalyst.
Signal Word
Warning
Hazard Statements
Precautionary Statements
Hazard Classifications
Skin Irrit. 2
Storage Class Code
13 - Non Combustible Solids
WGK
WGK 3
Flash Point(F)
Not applicable
Flash Point(C)
Not applicable
Certificates of Analysis (COA)
Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.
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Journal of the American Chemical Society, 7, 7245-7246 (1995)
American journal of physiology. Cell physiology, 302(12), C1741-C1750 (2012-03-31)
When skeletal muscle is stretched or injured, satellite cells, resident myogenic stem cells positioned beneath the basal lamina of mature muscle fibers, are activated to enter the cell cycle. This signaling pathway is a cascade of events including calcium-calmodulin formation
The Journal of Organic Chemistry, 64, 6462-6467 (1999)
The Journal of toxicological sciences, 37(2), 447-453 (2012-04-03)
Liver toxicity is a side effect observed with some herbal treatments, including Piper methysticum. The possible mechanisms responsible include inflammation subsequent to activation of liver macrophages and oxidative damage. Hepatotoxicity of the pharmacologically active component of Piper methysticum (kavalactones) was
Naunyn-Schmiedeberg's archives of pharmacology, 386(3), 255-264 (2012-12-12)
The present study was designed to investigate the potential of gadolinium, a stretch-activated calcium channel blocker in ischemic reperfusion (I/R)-induced brain injury in mice. Bilateral carotid artery occlusion of 12 min followed by reperfusion for 24 h was given to induce cerebral
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