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化驗
≥98% (HPLC)
形狀
powder
顏色
peach to light pink
溶解度
DMSO: ≥20 mg/mL
儲存溫度
2-8°C
SMILES 字串
Cc1[nH]c2ccccc2c1CCNC(=O)c3cccs3
InChI
1S/C16H16N2OS/c1-11-12(13-5-2-3-6-14(13)18-11)8-9-17-16(19)15-7-4-10-20-15/h2-7,10,18H,8-9H2,1H3,(H,17,19)
InChI 密鑰
ACAKNPKRLPMONU-UHFFFAOYSA-N
生化／生理作用
CK-636 结合在 Arp2 和 Arp3 之间，似乎阻碍 Arp2 和 Arp3 进入其活性构象。CK-636 插入 Arp3 的疏水核心并改变其构象。这两类化合物均抑制李斯特菌形成肌动蛋白丝彗尾和单核细胞形成伪足。
Charlotte Ford et al.
PLoS pathogens, 14(5), e1007051-e1007051 (2018-05-05)
Pathogens hijack host endocytic pathways to force their own entry into eukaryotic target cells. Many bacteria either exploit receptor-mediated zippering or inject virulence proteins directly to trigger membrane reorganisation and cytoskeletal rearrangements. By contrast, extracellular C. trachomatis elementary bodies (EBs)
Liang Ma et al.
Cell research, 25(1), 24-38 (2014-10-25)
Cells communicate with each other through secreting and releasing proteins and vesicles. Many cells can migrate. In this study, we report the discovery of migracytosis, a cell migration-dependent mechanism for releasing cellular contents, and migrasomes, the vesicular structures that mediate
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