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L3289

Sigma-Aldrich

LYSIS SOLUTION FOR BLOOD

sufficient for 100 reactions, for molecular biology

Synonyme(s) :

Blood direct PCR, Blood lysis buffer, Extract-N-Amp blood direct PCR lysis solution

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About This Item

Code UNSPSC :
12352200
Nomenclature NACRES :
NA.25

Qualité

for molecular biology

Forme

liquid

Utilisation

sufficient for 100 reactions

Température de stockage

−20°C

Catégories apparentées

Description générale

Lysis solution for blood in combination with neutralization solution for blood (N9784) aids in rapid extraction and neutralization of DNA from whole blood, whole blood dried on a blood card, and cultured mammalian cells.

This lysis solution is a component of the Extract-N-Amp™ and the REDExtract-N-Amp™ Blood PCR Kits

Application

Lysis solution for blood has been used for lysis of blood cells for DNA extraction prior to polymerase chain reaction (PCR) amplification. It has also been used for the isolation of genomic DNA for CRISPR-Cas9 gene editing.

Informations légales

Extract-N-Amp is a trademark of Sigma-Aldrich Co. LLC
REDExtract-N-Amp is a trademark of Sigma-Aldrich Co. LLC

Produit(s) apparenté(s)

Réf. du produit
Description
Tarif

XNAB

REDExtract-N-Amp™ Blood PCR Kit, sufficient for 100 extractions, sufficient for 100 amplifications

XNABR

REDExtract-N-Amp™ Blood PCR Kit, sufficient for 1000 extractions, sufficient for 1000 amplifications

XNAB2

Extract-N-Amp™ Blood PCR Kit, sufficient for 100 extractions, sufficient for 100 amplifications

XNABE

REDExtract-N-Amp™ Blood PCR Kit, sufficient for 100 extractions, sufficient for 500 amplifications

XNAB2RE

Extract-N-Amp™ Blood PCR Kit, sufficient for 1000 extractions, sufficient for 5000 amplifications

XNABS

REDExtract-N-Amp™ Blood PCR Kit, sufficient for 10 extractions, sufficient for 10 amplifications

P8240

REDExtract-N-Amp™ PCR ReadyMix™ for Blood, 12 mL sufficient for 1000 amplifications

P8115

Extract-N-Amp™ PCR ReadyMix™ for Blood, 12 mL sufficient for 1000 amplifications

N9784

Neutralization Solution for Blood, sufficient for 100 reactions, sufficient for 1000 reactions, for molecular biology

Pictogrammes

Corrosion
GHS05

Mention d'avertissement

Danger

Mentions de danger

H290,H314

Classification des risques

Eye Dam. 1 - Met. Corr. 1 - Skin Corr. 1B

Code de la classe de stockage

8A - Combustible corrosive hazardous materials

Classe de danger pour l'eau (WGK)

WGK 3

Point d'éclair (°F)

Not applicable

Point d'éclair (°C)

Not applicable

Certificats d'analyse (COA)

Recherchez un Certificats d'analyse (COA) en saisissant le numéro de lot du produit. Les numéros de lot figurent sur l'étiquette du produit après les mots "Lot" ou "Batch".

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Consulter la Bibliothèque de documents

Identification by random mutagenesis of functional domains in KREPB5 that differentially affect RNA editing between life cycle stages of Trypanosoma brucei
McDermott S M, et al.
Molecular and Cellular Biology, MCB-00790 (2015)
Identification by random mutagenesis of functional domains in KREPB5 that differentially affect RNA editing between life cycle stages of Trypanosoma brucei
McDermott S M, et al.
Molecular and cellular biology, MCB-00790 (2015)
Shuhan Chen et al.
Stem cell research, 45, 101804-101804 (2020-04-28)
Mutations in the Leucine rich repeat kinase 2 (LRRK2) gene are found in both familial and sporadic Parkinson's disease (PD), and are also associated with immune-related disorders including Crohn's disease (CD) and leprosy. We have generated two homozygous LRRK2 knockout
Meng Zhang et al.
Stem cell research, 41, 101602-101602 (2019-11-08)
Loss of function mutations in PARK2 (encoding PARKIN) cause autosomal recessive Parkinson's disease (PD), which often manifests at a juvenile age. Molecular and biochemical studies show that PARKIN functions as an E3 ubiquitin ligase controlling mitochondrial homeostasis. Yet, the exact
Huiling Wu et al.
The Journal of clinical investigation, 117(10), 2847-2859 (2007-09-15)
Ischemia/reperfusion injury (IRI) may activate innate immunity through the engagement of TLRs by endogenous ligands. TLR4 expressed within the kidney is a potential mediator of innate activation and inflammation. Using a mouse model of kidney IRI, we demonstrated a significant
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