Direkt zum Inhalt
Merck
Alle Fotos(2)

Dokumente

343226

Sigma-Aldrich

Nickelsulfid

99.7% trace metals basis, −150 mesh

Synonym(e):

Nickelsubsulfid

Anmeldenzur Ansicht organisationsspezifischer und vertraglich vereinbarter Preise
Alle Fotos(2)

About This Item

Empirische Formel (Hill-System):
Ni3S2
CAS-Nummer:
12035-72-2
Molekulargewicht:
240.21
EG-Nummer:
234-829-6
MDL-Nummer:
MFCD00217044
UNSPSC-Code:
12161600
PubChem Substanz-ID:
24861133
NACRES:
NA.23

Assay

99.7% trace metals basis

Form

powder

Eignung der Reaktion

reagent type: catalyst
core: nickel

Partikelgröße

−150 mesh

Dichte

5.8 g/mL at 25 °C (lit.)

Anwendung(en)

battery manufacturing

SMILES String

[Ni]=S=[Ni]=S=[Ni]

InChI

1S/3Ni.2S

InChIKey

YGHCWPXPAHSSNA-UHFFFAOYSA-N

Allgemeine Beschreibung

Major phase is Ni3S2. Product may also include a small amount of sulfur-rich phase. Nickel Sulfide (Ni3S2) exists as a heazlewoodite mineral in nature. The structure of Ni3S2 is hombohedral. Raman spectral analysis of Ni3S2 has been reported.

Anwendung

Nickel sulfide was implanted in mice, to induce the growth of malignant fibrous histiocytomas.

Verpackung

Packaged in glass bottles

Signalwort

Danger

Gefahreneinstufungen

Acute Tox. 3 Inhalation - Aquatic Acute 1 - Aquatic Chronic 1 - Carc. 1A Inhalation - Muta. 2 - Skin Sens. 1 - STOT RE 1 Inhalation

Zielorgane

Lungs

Lagerklassenschlüssel

6.1D - Non-combustible acute toxic Cat.3 / toxic hazardous materials or hazardous materials causing chronic effects

WGK

WGK 3

Flammpunkt (°F)

Not applicable

Flammpunkt (°C)

Not applicable

Persönliche Schutzausrüstung

Eyeshields, Faceshields, Gloves, type P3 (EN 143) respirator cartridges

Analysenzertifikate (COA)

Suchen Sie nach Analysenzertifikate (COA), indem Sie die Lot-/Chargennummer des Produkts eingeben. Lot- und Chargennummern sind auf dem Produktetikett hinter den Wörtern ‘Lot’ oder ‘Batch’ (Lot oder Charge) zu finden.

Besitzen Sie dieses Produkt bereits?

In der Dokumentenbibliothek finden Sie die Dokumentation zu den Produkten, die Sie kürzlich erworben haben.

Die Dokumentenbibliothek aufrufen

Kunden haben sich ebenfalls angesehen

Slide 1 of 6

1 of 6

Nickel(II)-sulfat anhydrous, 99.99% trace metals basis

Sigma-Aldrich

656895

Nickel(II)-sulfat

Nickel(II)-oxid 99.99% trace metals basis

Sigma-Aldrich

203882

Nickel(II)-oxid

Nickel powder, <1 μm, 99.8% trace metals basis

Sigma-Aldrich

268283

Nickel

Nickel powder, <50 μm, 99.7% trace metals basis

Sigma-Aldrich

266981

Nickel

Nickel nanopowder, <100 nm avg. part. size, ≥99% trace metals basis

Sigma-Aldrich

577995

Nickel

Molybdän(IV)-sulfid powder, <2 μm, 98%

Sigma-Aldrich

234842

Molybdän(IV)-sulfid

Joseph R Landolph et al.
Environmental health perspectives, 110 Suppl 5, 845-850 (2002-11-12)
In the past, exposure of workers to mixtures of soluble and insoluble nickel compounds by inhalation during nickel refining correlated with increased incidences of lung and nasal cancers. Insoluble nickel subsulfide and nickel oxide (NiO) are carcinogenic in animals by
M Okamoto
Differentiation; research in biological diversity, 61(5), 285-292 (1997-08-01)
It is well known that urodeles have the most powerful regenerative capacities among vertebrates, but there is little realisation that they are extremely resistant to spontaneous or chemically induced tumours. Regeneration and carcinogenesis have been considered to be two sides
Aaron Barchowsky et al.
The Journal of biological chemistry, 277(27), 24225-24231 (2002-04-30)
Inhalation of particulate nickel subsulfide (Ni3S2) causes chronic active inflammation and fibrosis of the lungs. However, the mechanisms for these effects are not well understood. Therefore, cell culture experiments with BEAS-2B human airway epithelial cells were conducted to test the
X Mao et al.
Cancer letters, 161(1), 57-62 (2000-11-18)
Differential display polymerase chain reaction (DD-PCR) was used to analyze the differentially expressed genes from nickel-transformed human embryonic lung (HEL) cells (MRC-9 and IMR-90) and their control counterparts (non-treated). Two genes, MS515 and IC82, were confirmed by Northern blot analysis.
Reactive oxygen-induced carcinogenesis causes hypermethylation of p16 (Ink4a) and activation of MAP kinase.
Govindarajan B, et al.
Molecular Medicine, 8(1) (2002)
Alle zugehörigen Dokumente anzeigen

Unser Team von Wissenschaftlern verfügt über Erfahrung in allen Forschungsbereichen einschließlich Life Science, Materialwissenschaften, chemischer Synthese, Chromatographie, Analytik und vielen mehr..

Setzen Sie sich mit dem technischen Dienst in Verbindung.