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EMU060971

Sigma-Aldrich

MISSION® esiRNA

targeting mouse Tmem132a

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About This Item

UNSPSC Code:
41105324
NACRES:
NA.51

description

Powered by Eupheria Biotech

product line

MISSION®

form

lyophilized powder

esiRNA cDNA target sequence

CCACCCTCTGGACTGCTAAGTTAGACCGCTTCAAGGGCTCAAAGCACCACACCAGCCTGATCACCTGTCACCGTGCTGGGCCTGCAGGGCCAGATTCCAGCCCCCTTGAACTGTCCGAGTTCCTGTGGGTGGACTTTGCAGTGGAGAACAGCACTGGTGGGGGTGTGGCAGTCACTCGCCCTGTCACGTGGCAGCTGGAGTACCCAGGTCAGGCCCCCGAAGCAGAGAAGGACAAAATGGTGTGGGAGATCCTGGTGTCTGAGCGGGACATCAGAGCCCTCATCCCTCTGGCCAAGGCTGAGGAGCTGGTGAACACGGCTCCATTGACTGGAGTGCCCCAGCGTATTCCGGTGCGGCTCGTCACTGTGGACAGTGGGGGAGCCTTGGAGGAGGTGACAGAGC

Ensembl | mouse accession no.

ENSMUSG00000024736

NCBI accession no.

NM_133804

shipped in

ambient

storage temp.

−20°C

Gene Information

mouse ... TMEM132A(98170) , Tmem132a(98170)

General description

MISSION® esiRNA are endoribonuclease prepared siRNA. They are a heterogeneous mixture of siRNA that all target the same mRNA sequence. These multiple silencing triggers lead to highly-specific and effective gene silencing.

For additional details as well as to view all available esiRNA options, please visit SigmaAldrich.com/esiRNA.

Legal Information

MISSION is a registered trademark of Merck KGaA, Darmstadt, Germany

Storage Class

10 - Combustible liquids

flash_point_f

Not applicable

flash_point_c

Not applicable

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Deng He et al.
International journal of molecular sciences, 16(7), 16313-16329 (2015-07-21)
The molecular events leading to nephrolithiasis are extremely complex. Previous studies demonstrated that calcium and transforming growth factor-β1 (TGF-β1) may participate in the pathogenesis of stone formation, but the explicit mechanism has not been defined. Using a self-created genetic hypercalciuric
Jingsheng Xia et al.
The Journal of physiology, 592(16), 3443-3461 (2014-05-27)
Store-operated calcium channels (SOCs) are calcium-selective cation channels that mediate calcium entry in many different cell types. Store-operated calcium entry (SOCE) is involved in various cellular functions. Increasing evidence suggests that impairment of SOCE is responsible for numerous disorders. A

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