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About This Item
Empirical Formula (Hill Notation):
C10H14N2
CAS Number:
Molecular Weight:
162.23
MDL number:
UNSPSC Code:
12352200
PubChem Substance ID:
form
ethanol solution
extent of labeling
60-87 Ci/mmol
shipped in
dry ice
storage temp.
−20°C
SMILES string
[H]CN1CCC[C@@H]1c2cccnc2
Gene Information
human ... CYP1A2(1544) , CYP2A6(1548)
mouse ... Cyp2a5(13087)
rat ... Chrna2(170945) , Chrnb2(54239)
Packaging
Packaged under Nitrogen
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Association of smoking status with health-related outcomes after percutaneous coronary intervention.
Jae-Sik Jang et al.
Circulation. Cardiovascular interventions, 8(5) (2015-05-15)
Patients who smoke at the time of percutaneous coronary intervention (PCI) would ideally have a strong incentive to quit, but most do not. We sought to compare the health status outcomes of those who did and did not quit smoking
Bo Zhang et al.
American journal of epidemiology, 181(7), 513-520 (2015-03-06)
In the present study, we examined the association between duration of nicotine replacement therapy (NRT) use and smoking cessation using data from the Ontario Tobacco Survey longitudinal study (3 waves of data collected between July 2005 and December 2009). We
Pek Kei Im et al.
Tobacco control, 24 Suppl 4, iv40-iv47 (2015-04-19)
To determine the most prominent individual and interpersonal triggers to quit smoking in China and their associations with sociodemographic characteristics. Data come from Waves 1-3 (2006-2009) of the International Tobacco Control (ITC) China Survey, analysed cross-sectionally as person-waves (N=14,358). Measures
Xiaosi Gu et al.
Proceedings of the National Academy of Sciences of the United States of America, 112(8), 2539-2544 (2015-01-22)
Little is known about how prior beliefs impact biophysically described processes in the presence of neuroactive drugs, which presents a profound challenge to the understanding of the mechanisms and treatments of addiction. We engineered smokers' prior beliefs about the presence
G K LeMasters et al.
The pharmacogenomics journal, 15(2), 189-195 (2014-08-27)
Cotinine is a proxy for secondhand smoke (SHS) exposure. Genetic variation along nicotine and cotinine metabolic pathways may alter the internal cotinine dose, leading to misinterpretations of exposure-health outcome associations. Caucasian children with available SHS exposure and hair cotinine data
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