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T0516

Sigma-Aldrich

Thromboxane B2

Synonym(s):

(5Z,9α,11RS,13E,15S)-9,11,15-Trihydroxythromboxa-5,13-dien-1-oic acid, TXB2

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About This Item

Empirical Formula (Hill Notation):
C20H34O6
CAS Number:
54397-85-2
Molecular Weight:
370.48
MDL number:
MFCD00135258
UNSPSC Code:
12352200
PubChem Substance ID:
24899894

assay

≥98% (HPLC)

form

powder

functional group

carboxylic acid

shipped in

ambient

storage temp.

−20°C

SMILES string

CCCCC[C@H](O)\C=C\[C@H]1OC(O)C[C@H](O)[C@@H]1C\C=C/CCCC(O)=O

InChI

1S/C20H34O6/c1-2-3-6-9-15(21)12-13-18-16(17(22)14-20(25)26-18)10-7-4-5-8-11-19(23)24/h4,7,12-13,15-18,20-22,25H,2-3,5-6,8-11,14H2,1H3,(H,23,24)/b7-4-,13-12+/t15-,16-,17-,18+,20?/m0/s1

InChI key

XNRNNGPBEPRNAR-JQBLCGNGSA-N

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Biochem/physiol Actions

Prostaglandin derivative that is released during anaphylaxis; induces arterial contraction and platelet aggregation.
Prostaglandin derivative that is released during anaphylaxis; induces arterial contraction and platelet aggregation. Review of endogenous thromboxane B2 in risk analysis for development of thrombogenic disorders.

pictograms

Health hazard
GHS08

signalword

Warning

hcodes

H361

Hazard Classifications

Repr. 2

Storage Class

11 - Combustible Solids

wgk_germany

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

ppe

Eyeshields, Gloves, type P3 (EN 143) respirator cartridges

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Certificates of Analysis (COA)

Lot/Batch Number
SLBD5303V
SLBC2766V
021M1501V
021M1501
020M1154

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European journal of pharmacology, 794, 234-245 (2016-11-30)
High on-aspirin platelet reactivity (HAPR) has been associated with compromised aspirin efficacy in patients with diabetes suffering from acute cardiovascular events, but the key mechanisms remain elusive. The objective of this study was to uncover the potential link between pathogenic
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BMC complementary and alternative medicine, 17(1), 396-396 (2017-08-12)
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C R Pace-Asciak et al.
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Early identification of infants at risk for complications from patent ductus arteriosus (PDA) may improve treatment outcomes. The aim of this study was to identify biochemical markers associated with persistence of PDA, and with failure of pharmacological treatment for PDA
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