857211
5-Bromo-4-chloro-3-indolyl β-D-galactopyranoside
98%
Synonym(s):
5-Bromo-4-chloro-3-indolyl β-D-galactoside, BCIG, X-gal
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About This Item
Empirical Formula (Hill Notation):
C14H15BrClNO6
CAS Number:
Molecular Weight:
408.63
Beilstein/REAXYS Number:
1402009
EC Number:
MDL number:
UNSPSC Code:
12352200
PubChem Substance ID:
assay
98%
form
powder
storage temp.
−20°C
SMILES string
OC[C@H]1O[C@@H](Oc2c[nH]c3ccc(Br)c(Cl)c23)[C@H](O)[C@@H](O)[C@H]1O
InChI
1S/C14H15BrClNO6/c15-5-1-2-6-9(10(5)16)7(3-17-6)22-14-13(21)12(20)11(19)8(4-18)23-14/h1-3,8,11-14,17-21H,4H2/t8-,11+,12+,13-,14-/m1/s1
InChI key
OPIFSICVWOWJMJ-AEOCFKNESA-N
Application
Histochemical substrate for β-galactosidase.
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Histochemical methods for acid beta-galactosidase: technics for semipermeable membranes.
Z Lojda
Histochemie. Histochemistry. Histochimie, 37(4), 375-378 (1973-12-31)
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PloS one, 8(3), e59195-e59195 (2013-03-26)
Our study had three objectives: to extend the plasmid-based transformation protocol to a clinical isolate of C. trachomatis belonging to the trachoma biovar, to provide "proof of principle" that it is possible to "knock out" selected plasmid genes (retaining a
Amy McCurley et al.
Nature medicine, 18(9), 1429-1433 (2012-08-28)
Hypertension is a cardiovascular risk factor present in over two-thirds of people over age 60 in North America; elevated blood pressure correlates with increased risk of heart attack, stroke and progression to heart and kidney failure. Current therapies are insufficient
Otso Häärä et al.
Development (Cambridge, England), 139(17), 3189-3199 (2012-07-27)
Uncovering the origin and nature of phenotypic variation within species is the first step in understanding variation between species. Mouse models with altered activities of crucial signal pathways have highlighted many important genes and signal networks regulating the morphogenesis of
Enzo R Porrello et al.
Proceedings of the National Academy of Sciences of the United States of America, 110(1), 187-192 (2012-12-19)
We recently identified a brief time period during postnatal development when the mammalian heart retains significant regenerative potential after amputation of the ventricular apex. However, one major unresolved question is whether the neonatal mouse heart can also regenerate in response
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